Increased expression of Myc‑interacting zinc finger protein 1 in APP/PS1 mice
Suppression of inflammation and acute lung injury by Miz1 via repression of C/EBP-δ | Nature Immunology
Frontiers | Identification of the SARS-CoV-2 Entry Receptor ACE2 as a Direct Target for Transcriptional Repression by Miz1
Identification of an atypical interaction site in the BTB domain of the MYC-interacting zinc-finger protein 1 - ScienceDirect
Miz-1 Activates Gene Expression via a Novel Consensus DNA Binding Motif | PLOS ONE
Suppression of inflammation and acute lung injury by Miz1 via repression of C/EBP-δ | Nature Immunology
Epithelial cell–specific loss of function of Miz1 causes a spontaneous COPD-like phenotype and up-regulates Ace2 expression in mice | Science Advances
Transcription Factor Miz-1 Is Required to Regulate Interleukin-7 Receptor Signaling at Early Commitment Stages of B Cell Differentiation - ScienceDirect
Increased expression of Myc‑interacting zinc finger protein 1 in APP/PS1 mice
The Interaction of Myc with Miz1 Defines Medulloblastoma Subgroup Identity - ScienceDirect
Biology | Free Full-Text | Myc-Interacting Zinc Finger Protein 1 (Miz-1) Is Essential to Maintain Homeostasis and Immunocompetence of the B Cell Lineage
Mice with lung epithelial cell-specific loss of function of Miz1 also... | Download Scientific Diagram
The Role of MIZ-1 in MYC-Dependent Tumorigenesis
Transcription Factor Miz-1 Is Required to Regulate Interleukin-7 Receptor Signaling at Early Commitment Stages of B Cell Differentiation - ScienceDirect
The role of MIZ-1 in MYC-dependent tumorigenesis. - Abstract - Europe PMC
Genetic disruption of the Miz1 POZ domain upregulates Cebpd... | Download Scientific Diagram
Frontiers | Identification of the SARS-CoV-2 Entry Receptor ACE2 as a Direct Target for Transcriptional Repression by Miz1
Epithelial cell–specific loss of function of Miz1 causes a spontaneous COPD-like phenotype and up-regulates Ace2 expression in mice | Science Advances
PDF) Epithelial cell–specific loss of function of Miz1 causes a spontaneous COPD-like phenotype and up-regulates Ace2 expression in mice
Biology | Free Full-Text | Myc-Interacting Zinc Finger Protein 1 (Miz-1) Is Essential to Maintain Homeostasis and Immunocompetence of the B Cell Lineage
Epithelial cell–specific loss of function of Miz1 causes a spontaneous COPD-like phenotype and up-regulates Ace2 expression in mice | Science Advances
Miz-1 Activates Gene Expression via a Novel Consensus DNA Binding Motif | PLOS ONE
Frontiers | Identification of the SARS-CoV-2 Entry Receptor ACE2 as a Direct Target for Transcriptional Repression by Miz1
