ALS-Linked SOD1 Mutant G85R Mediates Damage to Astrocytes and Promotes Rapidly Progressive Disease with SOD1-Containing Inclusions: Neuron
SOD1-G93A Mouse Models - QPS Neuropharmacology
Sagittal brain slices from a SOD1 G93A transgenic mouse at terminal... | Download Scientific Diagram
Neuron-Specific Expression of Mutant Superoxide Dismutase Is Sufficient to Induce Amyotrophic Lateral Sclerosis in Transgenic Mice | Journal of Neuroscience
Tempol improves neuroinflammation and delays motor dysfunction in a mouse model (SOD1G93A) of ALS | Journal of Neuroinflammation | Full Text
Figure 4 from Mutant superoxide dismutase 1 (SOD1), a cause of amyotrophic lateral sclerosis, disrupts the recruitment of SMN, the spinal muscular atrophy protein to nuclear Cajal bodies. | Semantic Scholar
IJMS | Free Full-Text | Abnormal Upregulation of GPR17 Receptor Contributes to Oligodendrocyte Dysfunction in SOD1 G93A Mice
Amyotrophic Lateral Sclerosis disease - QPS Austria
Figure 3.1 from The neuromuscular transmission of the SOD 1 ( G 93 A ) mouse model of Amyotrophic Lateral Sclerosis | Semantic Scholar
Overexpression of CCS in G93A-SOD1 mice leads to accelerated neurological deficits with severe mitochondrial pathology | PNAS
Wild-Type Nonneuronal Cells Extend Survival of SOD1 Mutant Motor Neurons in ALS Mice | Science
![PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar](https://d3i71xaburhd42.cloudfront.net/cf77f873405b248b7c9948af453d095fddb3ca81/3-Figure2-1.png "PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar")
PDF] Wild-type SOD1 overexpression accelerates disease onset of a G85R SOD1 mouse. | Semantic Scholar
004435 - SOD1-G93A Strain Details
Generation and analysis of innovative genomically humanized knockin SOD1, TARDBP (TDP-43), and FUS mouse models - ScienceDirect
Frontiers | Neuroprotective Effects of Shenqi Fuzheng Injection in a Transgenic SOD1-G93A Mouse Model of Amyotrophic Lateral Sclerosis
Guanabenz Treatment Accelerates Disease in a Mutant SOD1 Mouse Model of ALS | PLOS ONE
SOD1-positive aggregate accumulation in the CNS predicts slower disease progression and increased longevity in a mutant SOD1 mouse model of ALS | Scientific Reports
Parkin is a disease modifier in the mutant SOD1 mouse model of ALS. - Abstract - Europe PMC
Disease progression in G93A-SOD1 transgenic mice is accelerated by... | Download Scientific Diagram
IJMS | Free Full-Text | Brain Protease Activated Receptor 1 Pathway: A Therapeutic Target in the Superoxide Dismutase 1 (SOD1) Mouse Model of Amyotrophic Lateral Sclerosis
SOD1 (71G8) Mouse mAb | Cell Signaling Technology
Transgenic mice expressing H46R/H48Q/H63G/H120G (Quad) mutant SOD1... | Download Scientific Diagram
General behavior of SOD1 G93A mice. (A) Body weight variations in... | Download Scientific Diagram
SOD1-G93A Mouse Models - QPS Neuropharmacology
SOD1/Rag2 Mice with Low Copy Number of SOD1 Gene as a New Long-Living Immunodeficient Model of ALS | Scientific Reports
Frontiers | Human Motor Neurons With SOD1-G93A Mutation Generated From CRISPR/Cas9 Gene-Edited iPSCs Develop Pathological Features of Amyotrophic Lateral Sclerosis
Frontiers | Skeletal Muscle-Restricted Expression of Human SOD1 in Transgenic Mice Causes a Fatal ALS-Like Syndrome
A New AAV10-U7-Mediated Gene Therapy Prolongs Survival and Restores Function in an ALS Mouse Model - ScienceDirect
Overexpression of MT-I in SOD1 G93A mice normalizes copper... | Download Scientific Diagram
